Download Pathology of the Ovary, Fallopian Tube and Peritoneum by Nafisa Wilkinson PDF

By Nafisa Wilkinson

As the second one quantity within the Essentials of Diagnostic Gynecological Pathology sequence backed by way of the British organization of Gynecological Pathologists, Pathology of the Ovary, Fallopian Tube and Peritoneum is without doubt one of the only a few dealing utterly with this topic.

Pathology of the Ovary, Fallopian Tube and Peritoneum introduces the subject with a dialogue of the anatomy, improvement, histology and general functionality of the ovary, through chapters on non-neoplastic issues of the ovary; surgical procedure and clinical administration of ovarian melanoma; and using imaging, frozen sections and cytology in ovarian pathology. The ebook then is going directly to describe particular issues of the ovary, fallopian tube and peritoneum intimately, and finishes with a bankruptcy on specimen cut-up.

Pathology of the Ovary, Fallopian Tube and Peritoneum has been written to be invaluable diagnostically to normal in addition to expert gynecological histopathologists and pathologists in education. Gynecologists, oncologists, genitourinary physicians and melanoma nurse experts will locate specialist insights the following that may assist in therapy and counselling in their patients.

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Extra info for Pathology of the Ovary, Fallopian Tube and Peritoneum

Sample text

Premature Ovarian Failure Premature ovarian failure (POF) is a good example of the failure of one or more aspects of ovarian function and highlights the multiple roles played by the ovary in maintaining normal physiology. , postpubertal) amenorrhea, which features premature follicle depletion or arrested folliculogenesis [271, 272]. , FSH/LH receptor and BMP-15 mutations), or idiopathic [273]. X chromosome abnormalities such as Turner syndrome account for the majority of primary amenorrhea cases associated with ovarian dysgenesis, and, predictably, these feature an absence of both pubertal development and its associated growth spurt [173, 274, 275].

Angiogenesis in reproductive physiology: follicular development, formation and maintenance of the corpus luteum. Ginecol Obstet Mex. 2002;70:17–27. 75. Indraccolo S. Interferon-alpha as angiogenesis inhibitor: learning from tumor models. Autoimmunity. 2010;43:244–7. 76. Połeć A, Ráki M, Åbyholm T, Tanbo TG, Fedorcsák P. Interaction between granulosa-lutein cells and monocytes regulates secretion of angiogenic factors in vitro. Hum Reprod. 2011;26:2819–29. 77. Fraser HM. Regulation of the ovarian follicular vasculature.

Although it has been proposed that this neuroendocrine anomaly could trace its origins to prenatal androgenization (consistent with elevated gestational androgen levels in PCOS and the fact that the disorder clusters in families in the absence of well-defined genetic causes), whether these variables are causative or coincidental remains to be proven [31, 282, 293, 294]. Regardless of the mechanism involved, the rapid GnRH pulsatility in PCOS and its resultant increased LH:FSH ratio translate into excessive ovarian androgen biosynthesis and ovulatory dysfunction, thereby establishing a self-perpetuating cycle of endocrine dysfunction.

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