Download Lewis's Child and Adolescent Psychiatry: A Comprehensive by Andres Martin, Fred R. Volkmar PDF

By Andres Martin, Fred R. Volkmar

Established for fifteen years because the typical paintings within the box, Melvin Lewis's Child and Adolescent Psychiatry: A accomplished Textbook is now in its Fourth variation. lower than the editorial course of Andrés Martin and Fred R. Volkmar—two of Dr. Lewis's colleagues on the world-renowned Yale baby learn Center—this vintage textual content emphasizes the connection among easy technological know-how and medical study and integrates medical ideas with the realities of drug interactions.

This variation has been reorganized right into a extra compact, clinically suitable ebook and entirely up to date, with two-thirds new contributing authors. the hot constitution comprises economics, range, and a heavy concentrate on evidence-based perform. a variety of new chapters comprise genetics, study method and records, and the continuum of care and location-specific interventions.

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Additional resources for Lewis's Child and Adolescent Psychiatry: A Comprehensive Textbook (4th Edition)

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Deletions of chromosome 15 as a cause of the Prader-Willi syndrome. N Engl J Med 304:325–329, 1981. Lifton RP: Molecular genetics of human blood pressure variation. Science 272:676–680, 1996. : Strong correlation of elastin deletions, detected by FISH, with Williams syndrome: Evaluation of 235 patients. Am J Hum Gen 57:49–53, 1995. Lubs H: A marker X chromosome. Am J Hum Gen 21:231–244, 1969. : Is Angelman syndrome an alternate result of del(15)(q11q13)? Am J Med Genet 28: 829–838, 1987. Martin JB: Molecular genetic studies in the neuropsychiatric disorders.

1995). , 1996). Tassabehji and colleagues (1996) were the first to report the deletion of a second gene in 20 out of 20 Williams syndrome patients. The sequence of this gene is nearly identical to a previously characterized gene called LIM kinase 1. , 1995), mutations in this gene are being searched for and are possible candidates responsible for the cognitive deficits seen in Williams syndrome patients. For a brief period, it was thought that elastin and LIM kinase 1 were the only genes within the deleted region.

5. Lissencephaly. The normal cerebral cortex is a highly organized structure, and its six layers are shown on the left (1–6). In contrast, the lissencephalic brain lacks the normal pattern of sulci and gyri, and there are only four layers. (Adapted with permission from Reiner O, Lombroso PJ: Lissencephaly. ) In 1993, a large deletion on chromosome 17 was found in two patients with MDS. , 1993). This gene and genes on either side of it were absent owing to the deletion and is another example of a contiguous gene syndrome.

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