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By W. Michael Scheld, James M. Hughes, Barbara E. Murray

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EPI Newsl. 23(4):2–3. 9. Robbins, F. , and C. A. de Quadros. 1997. Certification of the eradication of indigenous transmission of wild poliovirus in the Americas. J. Infect. Dis. 175(Suppl. 1):281–285. 10. Wood, D. , R. W. Sutter, and W. R. Dowdle. 2001. Stopping poliovirus vaccination after eradication: issues and challenges. Bull. W. H. O. 78:347–363. 11. World Health Organization. 1998. Report of the Meeting on the Scientific Basis for Stopping Polio Immunization. World Health Organization, Geneva, Switzerland.

Rawlinson. 2001. Hepatitis and death following vaccination with 17D-204 yellow fever vaccine. Lancet 358:121–122. 10. Freedman, D. , P. E. Kozarsky, L. H. Weld, and M. S. Cetron. 1999. GeoSentinel: the global emerging infections sentinel network of the international society of travel medicine. J. Travel Med. 6:94–98. 11. , K. V. Pugachev, C. L. Santos, S. W. Ochran, A. V. Jabor, S. G. Rodrigues, R. S. Marchevsky, M. S. Friere, L. F. C. Almeida, A. C. R. Cruz, A. M. Y. Yamamura, I. M. Rocco, E. S.

Of the two persons who were serologically tested, one had unusually high levels of yellow fever antibodies (1:10,240 titer) (Werfel and Popp, letter, 2001). Whether and in what way underlying genetic or acquired host factors may have contributed to the course or outcome of yellow fever vaccine-associated viscerotropic disease are unknown. AGE-ASSOCIATED RISK Accurately measuring the incidence of vaccine-associated viscerotropic and neurotropic disease is difficult because adequate prospective data are unavailable.

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